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With regard to muscle atrophy in cachexia, studies have focused on different cell signaling pathways, including the myostatin pathway, TNF- pathway, TGF/p38/MAPK pathway, NFB pathway, IL-6 pathway [12], Notch/-catenin pathway, corticosteroid pathway, IGF/Akt pathway [54], and FOXO1 /FOXO3a pathway [55], among others

By contrast, skeletal muscle metabolites at rest did not significantly differ between the groups

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Long-chain fatty acyl-CoA thioesters cannot enter the mitochondrial matrix in animals because they are not able to pass through the inner mitochondrial membrane