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Senescence-associated heterochromatic foci formation and nuclear DNA alteration in senescent astrocytes results in attenuated expression of genes related to cell proliferation, expression of DNA damage response (DDR) markers, as well as increased nuclear size (Souza et al., 2015

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However, under pathological conditions such as chronic infections, metabolic dysregulation, or age-related immune decline, senescent cells persist beyond their beneficial window