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In vivo oxidation of tyrosine induced by biologically important radicals, UV radiation or enzymes, produces a wide range of oxidized products whose formation significantly depends on the nature of the oxidant and the environment of the residue 6,7,8

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Synaptic alterations are a hallmark of numerous neurological and psychiatric disorders, making the restoration of synaptic efficacy a central therapeutic goal [203, 204]

We propose that comparable approaches aimed at developing agents targeting IGF-1R downstream signaling molecules specifically implicated in each type of cancer on a case-by-case basis could have better anticancer resistance effects with less toxicity

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