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Increased manganese uptake by primary astrocyte cultures with altered iron status is mediated primarily by divalent metal transporter

littoralis cytosolic GSTs in terms of their reactivity with ITCs

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These substances include bacterial endotoxins (particularly lipopolysaccharide, LPS), whole bacteria, and damage-associated molecular patterns (DAMPs) released from injured intestinal cells

What follows is theoretical framework based on individual compound dosing data and pharmacological principles