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As gut microbiota are highly responsive to diet, and nutritional support is already a protocolized part of TBI care (due to observational studies demonstrating an association between delayed enteral nutrition and worse neurologic outcome [113]) there has been much interest in identifying dietary interventions which can improve recovery and reduce mortality [114]

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Proteomic analyses further suggest that gene expression changes related to oxidative stress (GPX1, superoxide dismutase 1), metabolism (ACACA, GAPDH), placentation (PGF, VEGF-A), and stem cell differentiation (POU5F1, SOX2) may underlie the observed improvement in embryo quality (Marinaro et al., 2018

Though the mechanism for these processes, remains unclear, it is hypothesized that restoration of genes in glucose/lipid metabolism, enzyme alterations in the liver, and rendering immunity or protecting inflammation in the cardiovascular system would be an additive contributor to this therapeutic approach

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