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Discussion The purpose of this study was to investigate long-term (over a year) changes in antioxidant levels following irradiation in vivo

MLKL drives basal resistance to ferroptosis by depleting PUFAs, whereas ACSL4 promotes basal resistance to necroptosis by making the membrane less susceptible to MLKL-driven permeabilization [16, 214]

Marotta A, Sarno E, Del Casale A, Pane M, Mogna L, Amoruso A, et al

Furthermore orexin A (5x10-11M, 10-10M and 10-9M), caused a significant increase in cell proliferation in dose-dependent manner (about 21%, 96% and 160%, respectively)