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Recent evidence suggests that MCC is more likely to originate from dermal fibroblasts, epidermal keratinocytes, or immature totipotent stem cells that undergo neuroendocrine differentiation during the process of malignant transformation [57, 58]

They regulate blood-brain exchanges [1] at a time when the cerebral vascular network is not fully developed and parenchymal blood flow is low [2, 3]

Levels of glutathione fluctuate due to nutrient concentrations, exercise, oxidative stress and ageing

Key cytoprotective enzymes and molecules involved in ROS scavenging, including superoxide dismutase (SOD), glutathione peroxidase (GPx), catalase (CAT), and glutathione (GSH), play central roles in maintaining redox homeostasis

The UPR-PERK pathway is not a promising therapeutic target for mutant SOD1-induced ALS

Bone marrow biopsy and aspiration after 1 week of intramuscular vitamin B 12 supplementation was negative for tuberculosis and atypical leukaemic cells with a normal karyotype on chromosome analysis