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This vulnerability stems partly from their antioxidant defensesparticularly in the liver and red blood cellsbeing somewhat more limited than those of other animals

demonstrated that QE reversed the inhibition of transcription factor EB (TFEB) nuclear translocation incited by ethanol and exhibited similar effect to Torin 1 mTOR activity inhibitor) which could promote TFEB nuclear translocation, indicating that QE could alleviate chronic ethanol consumption induced liver injury and autophagic flux suppression by regulating mTOR-TFEB pathway (Li et al., 2019)

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doi: 10.1111/j.1365-2036.2006.03172.x

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