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41 (Table 1, Supplementary Table 1)

Preclinical studies in in vitro models have shown that simvastatin exerted its anti-tubercular activity through cholesterol-driven autophagy, which is mediated by the adenosine monophosphate-activated protein kinase (AMPK)mammalian target of the rapamycin complex 1 (mTORC1)TFEB axis [58] (Fig

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These results indicate that CSE treatment did not significantly impact barrier integrity, but led to increased cytotoxicity, elevated inflammatory markers, and heightened oxidative stress

Clinical research suggests that long-term use of lithium may cause hypercalcemia in 10% to 60% of patients