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Presynaptic kainate receptors that enhance the release of GABA on CA1 hippocampal interneurons

And I think that understanding the biology of what ApoE4 is doing because now there's research, a lot of it coming out of UCSF Gladstone Institute showing that in addition to a loss of function with the ApoE4 allele, there's also a dominant negative effect

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The restoration of spatial learning in APP/PS1 transgenic mice, which develop amyloid plaques and cognitive deficits analogous to human Alzheimer pathology, suggests that massive synaptogenic stimulation can override the connectivity losses caused by amyloid accumulation

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