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To date, it has been observed that the treatment with this kind of antitumor agents causes ROS generation, mitochondrial dysfunction (including frataxin deficiency, mitochondrial DNA damage and defective components of ETC), loss in antioxidant enzymes, ion channels disturbances and nerve tissue damage (protein carbonylation and lipid peroxidation) (Areti et al., in vitro and in vivo biochemical effects after exposure to high concentrations of oxaliplatin, including neuronal activation of the purinoreceptor subtype 7, ROS and NO production, lipid peroxidation, loss of mitochondrial transmembrane potential and further apoptosis via caspase 3 activation

Vitamin C, collagen solution, growth factors, and CoQ10 stimulate collagen production, improving firmness and reducing fine lines

Fernblock is an excellent candidate and example of specific compounds researched from extract-to-pill-to-patient, which provides enormous benefits in reducing risks for neoplasm and protecting from UV

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In a cross-over design, patients switched between placebo and EGCG treatment after 6 months, allowing further assessment of EGCG's effects

The p53-inducible apoptotic protein AMID is not required for normal development and tumor suppression