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In this context, ROS-triggered TP53 activation heightens mitochondrial membrane permeability, leading to the release of pro-apoptotic factors from mitochondria

Vandrey, R., Raber, J

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Sestrins were the first identified amino acid sensors in response to cytosolic leucine levels

Transient induction of antioxidant enzymes and nitric oxide synthase were also delayed or repressed during the regenerative response

We have generated the McGill-R-Thy1-APP transgenic (Tg) rat model exhibiting an AD-like amyloid pathology with a prolonged pre-plaque stage, which allows for studying the effects of gradual iA accumulation well before extracellular plaque formation 12,14,36