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DZN-induced AChE inhibition results in acetylcholine accumulation, overstimulating muscarinic and nicotinic receptors

To sustain continuous urine production, every cell in the kidneys undergoes metabolic reprogramming to meet energy demands, particularly in the early stages of DKD

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at higher molar ratios, it favors Cu 2+ -induced aggregates, which hinder fibrillization (100)