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Furthermore, therapeutic strategies using glucagon receptor antagonists to reduce high blood glucose in diabetics need to be reassessed, as they may negatively impact the function of the cells that produce insulin, the hormone diabetic patients need the most. Although their translational research involved both human tissue and mice, the researchers emphasized one uniquely human point

Background Cardiovascular disease (CVD) is a leading cause of mortality worldwide and it produces major burdens on morbidity, quality of life, and social costs [1]

In natural killer T-cell lymphoma (NKTCL), blocking SLC1A1-mediated glutamine addiction in tumor cells induced PD-L1 expression and inhibited CD8(+) T-cell activity 149

We introduced Dox-inducible FL Flag-tagged p21, or isolated N and C terminal truncation mutants (p21 N and p21 C , respectively), into HT-1080 p21 KO cells (Tarangelo et al, 2018)

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When HK-2 cells were treated with MLN4924, the UBE2m overexpression-induced increase in neddylation and expression of NEDD4 was rescued, as was the decrease in PPAR expression induced by NEDD4 elevation (Fig