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and after intracerebral hemorrhage, clathrin acid improves ferroptosis through cysteine 66 modification of the GPx4 site after intracerebral hemorrhage, alteration enhances its enzymatic activity by alkylation modification, thereby inhibiting neuronal ferroptosis, and the Irg1/cleroconanic acid/GPx4 axis may be a future therapeutic strategy to protect neurons from ferroptosis after intracerebral hemorrhage

Chemoattractant CCL2 contributes to metastasis in different types of cancers

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