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For instance, how Nrf2, Sirt1, and Sirt3 synergistically or antagonistically modulate GPX4/FTH1 expression under chronic high-glucose conditions remains undefined

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Since selenium supplementation in the forms of sodium selenite and SeM was well documented to induce the expressions of seleno-GPxs through their metabolic assimilation into bioavailable selenium (hydrogen selenide), the initial studies focused on exploring selenium supplementation as a strategy to achieve protection against radiation-induced toxicity [38, 66]

Additionally, our molecular experiments confirmed that chemotherapy drugs increase the expression levels of intracellular GSH and antioxidant enzymes, leading to chemotherapy resistance during neoadjuvant chemotherapy

Splenectomy has been the primary mode of therapy

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