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Indeed, EGR-1 was found to contribute to the pathogenesis of atherosclerosis [123], stenosed calcific valvular disease [124], and cardiac hypertrophy [125,126]

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It improved escape latency only in males, and only temporarily (Trial 2)

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[3] Seiwerth S, et al

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Its function involves interacting with the central nervous system to promote deep sleep while also influencing various physiological processes

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[DOI] [PubMed] [Google Scholar] 45.Sorenson J.R., Soderberg L.S., Chidambaram M.V., de la Rosa D.T., Salari H., Bond K., Kearns G.L., Gray R.A., Epperson C.E., Baker M.L

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