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Additionally, signaling pathways such as PI3K/Akt/mTOR and metabolites like succinate can also regulate the expression and stability of HIF-1 (71, 72)

Individuals who are homozygous for the null-GSTM1 or null-GSTT1 genotypes lack the respective enzyme functions (Seidegaard et al, 1986

Whether partial TCA cycle reverse activity due to the large accumulation of fumarate, leading to SDH reversal and fumarate reduction to succinate, as it has been observed in ischaemia [36], or cells treated with mitochondrial inhibitors [37], occurs in Fh -1 deficient cells has not been reported

27, 29 TxNIP can also function through alternative mechanisms such as Jun activating binding protein affecting its influence on p27 kip1 [ref

Vashaghian M, Diedrich CM, Zandieh-Doulabi B, Werner A, Smit TH, Roovers JP