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Functional crosstalk among oxidative stress and O-GlcNAc signaling pathways

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It also suppresses NF-kB activation and modulates macrophage polarization toward an M2 anti-inflammatory phenotype

Beta-tubulin and beta-actin expression was significantly suppressed while vimentin levels were elevated in mice lacking VAT resident macrophages and mice deficient in myeloid SerpinB2 (Fig

Recent studies have suggested that lysosomal dysfunction in SCs is linked to the pathophysiology of BIPN (Wu et al., 2023)