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Moreover, due to augmentation of GPX4 binding sites provided by the formed nanostructure, Gi-F exhibits increased inhibition of GPX4 activity by assembly enhanced binding ( AEB ) effect in cells, increasing the reactive oxidative species (ROS) produced by FeTCPP-based Fenton reaction

To view a copy of this licence, visit About this article Cite this article Devaux, A., Boucher, D., Villger, R

In MDA-MB-231 cells, CuPDTC induces calpain-dependent PARP1 cleavage and apoptosis, and this process is reversed by calpastatin (a calpain inhibitor) (Milacic et al., 2008)