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COX Members and Metabolites in Atherosclerosis The potent regulatory effects of AA and its derivatives on inflammation and coagulation indicate their critical roles in the occurrence and development of atherosclerosis

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Three main mechanisms leading to hyperhomocysteinemia are these: (1) an inhibition of the homocysteine remethylation pathway dependent on folate or cobalamin deficiency, on the C677T mutation of MTHFR, or on the functional deficiency in methionine synthase