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Taken together, these results suggested that redox control pathways in subcellular compartments are separately and precisely controlled to meet the specific redox requirements in these distinct locations.

All u SOD1 variants had K D values greater than u SOD1 WT (9 1 nM), implying that these single residue substitutions are sufficient to disturb the quaternary structure (Table 1)

Glutamate-cysteine ligase protects cells from ferroptosis by decreasing glutamate levels, converting them to -glutamyl peptides, and boosting GSH production (36) ( Figure 1 )

Hirsch FR, Varella-Garcia M, Bunn PA, Di Maria MV, Veve R, Bremnes RM, et al

Edge-site engineering of defective Fe-N 4 nanozymes with boosted catalase-like performance for retinal vasculopathies