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Tau in neurodegenerative diseases: Molecular mechanisms, biomarkers, and therapeutic strategies

10.1016/j.atherosclerosis.2008.07.010 118

Importantly, damage-associated molecular patterns (such as mitochondrial DNA, fibronectin extra domain A, and galectin-3) released during tissue damage may activate NETs through interaction with neutrophil surface proteins [172]

The ability of quercetin to suppress the production of pro-inflammatory cytokines is mediated by its suppression of the activation of ERK and p38 MAP kinase and NF-B/IB signal transduction pathways (Tang et al., 2019)

doi: 10.1002/ddr.21557 186 WangCHeCLuSWangXWangLLiangSet al

Dis., 133: 191-196