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DIM has been shown to reduce DNA hypermethylation of CpG islands (hallmark feature of cancer activity), reduce intestinal inflammation, function to mildly inhibit aromatase, and enhance DNA repair mechanisms

No enhancement or reduction of the anti-tumor activity of doxorubicin or etoposide was seen

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RSL3 induces DNA damage, inhibits DNA repair, and promotes autophagy in PTC cells We showed that the RSL3-mediated GPX4 inhibition induced multi-fold rise in intracellular ROS in PTC cells in vitro

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Genetic and biochemical data have shown that LRRK2 pathogenic variants particularly Gly2019Ser and variants in the GTPase Roc and COR domains of the protein cause a toxic, gain-of-function-mechanism mediated hyperactivity of LRRK2 kinase (Chan et al