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Nada SE, Williams FE, Shah ZA (2016) Development of a novel and robust pharmacological model of okadaic acid-induced Alzheimers disease in zebrafish

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187,188,189 In CCl 4 -induced liver injury, deletion of YAP/TAZ in BECs hinders the recruitment of phagocytic macrophages, impairing necrotic cell clearance and ultimately leading to regeneration defects at injury sites

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It stimulates glucose uptake, increases fat metabolism, and boosts mitochondrial function

Demyanenko S, Gantsgorn E, Rodkin S, Sharifulina S