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RIP1 autophosphorylation is promoted by mitochondrial ROS and is essential for RIP3 recruitment into necrosome

doi: 10.1002/hep4.1728 211 LuXLiuLShanWKongLChenNLouYet al

In contrast, survival rates were similar between vector-control mice and those expressing the loss-of-function mutated hepsin, with all mice in these groups succumbing within 30 h (Fig