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Given that the pathogenesis of antiretroviral toxic neuropathy is based on the disruption of mitochondrial energy supply one can assume that the clinical effect of ALCAR is dependent on its mitochondrial availability

In: Kumar N (ed) Mercury Toxicity Mitigation: Sustainable Nexus Approach

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Q2: What is the advantage over Cyanocobalamin

Biochemical Test and ELISA Serum of DILI and healthy controls were collected and frozen at -80 for the subsequent biochemical or ELISA measurement