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Results were reported as pooled hazard ratios (HR) with 95% confidence intervals (CI) for the association of NAFLD, with and without advanced fibrosis, with fatal, non-fatal, or combined CVD events

doi: 10.3390/antibiotics13040334 63 HerreroMCifuentesAIbaezE

In CKD, uremic toxin accumulation (e.g., indoxyl sulfate, acrolein), impaired antioxidant defense systems, and persistent inflammation collectively promote excessive production of reactive oxygen species (ROS), which disrupt erythrocyte membrane integrity, activate Ca 2+ -permeable cation channels, and trigger downstream eryptotic events (e.g., phosphatidylserine (PS) externalization, cell shrinkage, and cytoskeleton degradation) (Table 2 provides a detailed overview of their mechanisms, effects, and supporting evidence

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Cytosolic Ca2+ signals can be viewed as the result of two opposing factors: influx into and efflux out of the cytosol (Sanders et al., 1999)