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doi: 10.1371/journal.pntd.0002514 [DOI] [PMC free article] [PubMed] [Google Scholar] Lundqvist J, Larsson C, Nelson M, Andersson M, Bergstrom S, and Persson C (2010)

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Although research on ferroptosis in lens diseases is still in its infancy, existing evidence have indicated that aged human lenses and cataractous lenses exhibit hallmarks of ferroptosis, including disrupted redox homeostasis, reduced GSH levels, impaired GPX activity, elevated redox-active iron content, and increased lipid peroxidation [26, 27]