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Redox-crippled MitoQ potently inhibits breast cancer and glioma cell proliferation: A negative control for verifying the antioxidant mechanism of MitoQ in cancer and other oxidative pathologies

It promotes fibroblast proliferation, stimulates collagen and elastin synthesis, activates antioxidant defense enzymes, and has documented anti-inflammatory effects

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In the conversion of methylmalonyl CoA to succinyl CoA, where adenosylcobalamin plays a fundamental role as a cofactor, there is a defect in this reaction if there is not enough vitamin B12