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Long-term or continuous use, especially at high doses, should be reviewed periodically by your doctor to avoid unnecessary exposure, potential nutrient imbalances, or interactions with other therapies

Further studies have shown that PKC directly phosphorylates Ser 40 within the Neh2 domain of NRF2, which is necessary for the dissociation of NRF2 from Keap1 [41]

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Role of New Biomarkers: Functional and Structural Damage

KEAP1-dependent NRF2 activators Electrophilic compounds The most known NRF2 activators are electrophiles electron-deficient species able to form covalent bonds with electron-rich nucleophiles through a variety of chemical pathways which can covalently bind and modify cysteine residues in KEAP1 [273, 274]

A considerable number of evidences provided by Sikiric et al