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The nonenzymatic functions of GSTs involve their interactions with cellular proteins, such as, Jun N-terminal kinase,(JNK), tumor necrosis factor receptor-associated factor-2 (TRAF2), apoptosis-signal-regulating kinase 1 (ASK), serine/threonine kinases (PKA, PKC), and tissue transglutaminase 2 (TGM2), during which, either the interacting protein partner undergoes functional alteration or the GST protein itself is post-translationally modified and/or functionally altered [53], [74]

By analogy, protein (de)-phosphorylation, albeit thermodynamically favorable, is not controlled by the G for ATP hydrolysis or Atkinson's highly quoted energy charge (Atkinson and Fall, Examples of redox-regulated pathways Many cellular functions have been already associated with redox regulation

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Regular IV therapy helps create a baseline of antioxidant strength

doi: 10.1093/pcp/pch008 54 OtulakK.GarbaczewskaG

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