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Conclusion: We report a national study of adult HCV nonviremic liver transplant recipients from HCV Ab+/NAT- donors with a HCV viremia rate of =4.5mmol/L)

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By integrating microtranscriptomic, Limited Proteolysis-Small Molecule Mapping (LiP-SMap), and small molecule-protein interaction analysis, we find that uridine directly targets poly(rC)-binding protein 1 (PCBP1), thereby suppressing ferroptosis, maintaining mitochondrial function, alleviating oxidative damage, and ultimately improving the quality of aged oocytes