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2) Defect in the melanin transfer between melanocytes and keratinocytes, resulting in pigmentary incontinence.[10] We consider that in present case the dominant mechanism of hyper pigmentation is not a defect in melanin transport but is rather an increase in melanin synthesis

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Although patients express satisfaction with and continue to receive treatment with botulinum toxin, our data are not supported by the compilation of a subjective questionnaire such as the SAQ, which is considered a reliable tool and useful in evaluating important sequelae, such as synkinesis

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doi:10.1177/0884533609332174

doi: 10.1039/C5BM00052A 78 FadeelB