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Although the precise mechanisms underlying KDs effects in PD remain incompletely understood, evidence suggests that the diet can modulate glucose utilization in the brain, neurotransmitter metabolism, insulin signaling, oxidative stress, neuroinflammation, and ion channel activity, particularly potassium channels [20]

A genome-wide RNA interference screen revealed siRNAs targeting CARS (cysteinyl-tRNA synthetase) as robust genetic suppressors of erastin-induced ferroptosis [37]

6-AH family members were found to act as mimics of the dimerization domain of HGF (hinge region), and inhibited the interaction of an HGF molecule with a 3 H-hinge region peptide resulting in an attenuated capacity of HGF to activate its receptor Met

We further tested whether the methylation of UBE2m affects NEDD4 neddylation using exogenous and endogenous assays

"Copper supplementation in humans does not affect the susceptibility of low density lipoprotein to in vitro induced oxidation (FOODCUE project)"

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