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BV has been shown to increase the expression and levels of several pro-apoptotic mediators, such as cytochrome C, Bcl-2-associated X protein (Bax), protein 53 (p53), Bcl-2 homologues antagonist/killer (Bak), caspase-3, caspase-9, and numerous death receptors while reducing the anti-apoptotic mediator Bcl-2 [37, 38, 52]

These studies provide examples in cancer models that the redox-regulating properties of certain selenoproteins could decrease pro-inflammatory eicosanoid production and reduce inflammatory-dependent tumour progression

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Blood monocytes: distinct subsets, how they relate to dendritic cells, and their possible roles in the regulation of T-cell responses