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The copper-mediated inhibition of PHD and the resulting enhancement of HIF-1 function and activity will enhance downstream transcription of HIF-1 targeted genes, leading to metabolic reprogramming that enables tumors to better adapt to hypoxic environments

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Furthermore, peripheral blood monocytes of human subjects with G6PD deficiency show reduced secretion of inflammatory cytokines such as TNF and IL-1 [121]

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