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L., Aramburu, O., Vzquez, C

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In the present study, we demonstrate that freshly isolated mitochondria rapidly depolarize upon exposure to colistin and that pharmacological inhibition of the opening of the mitochondrial permeability transition pore (mPTP) protected proximal tubular cells from colistin-induced cytotoxicity in vitro and reduced the risk of kidney injury in mice

[1] ALCAR modulates phospholipid metabolism and synaptic morphology while enhancing synthesis of neurotrophic factors and neurotransmitters

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