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Systematic analysis of human cells lacking ATG8 proteins uncovers roles for GABARAPs and the CCZ1/MON1 regulator C18orf8/RMC1 in macroautophagic and selective autophagic flux

These inflammatory molecules included eotaxin, granulocyte colony-stimulating factor (G-CSF), granulocytemacrophage colony-stimulating factor (GM-CSF), interferon (IFN)-, keratinocyte chemoattractant (KC), monocyte chemoattractant protein (MCP)-1, macrophage inflammatory protein (MIP)-1, MIP-1, regulated on activation, normal T cell expressed and secreted (RANTES), tumor necrosis factor (TNF)-, and interleukin (IL)-1, -1, -2, -3, -4, -5, -6, -9, -10, -12(p40), -12(p70), -13, and -17A

However, the balance in HCC is disordered due to excessive angiogenic factors that are secreted by tumor cells, endothelial cells and pericytes

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