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Macrophage polarization-related biomarkers in ovarian cancer The traditional M1/M2 paradigm suggests that M1 macrophages, induced by IFN-, TNF-, and TLR signaling, promote anti-tumor immunity through the production of pro-inflammatory cytokines and reactive oxygen species

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These activated T helper cells then release interleukins, specifically IL-4 and IL-5, which stimulate the MMCs to produce more of the mast cell growth factor IL-9