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Molecular substrates of schizophrenia: homeostatic signaling to connectivity

147 Ramos-ZaldvarH

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Methylation can be impaired because SAM production is inadequate, because SAH accumulates and inhibits methyltransferase enzymes, or because both problems occur together

Thus, not only PGC 1 upregulation is paralleled by an improvement in insulin sensitivity, but also PGC 1-dependent defects in mitochondrial oxidative metabolism are reversible confirming this transcription cofactor as a valuable target to restore insulin sensitivity and the aberrant transcriptional control of mitochondrial biogenesis (Mootha et al., 2003