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Phosphatase and tensin homolog (PTEN) prevents G6PD from activation via the PTEN/Tcl1/hnRNPK/G6PD axis (61)

This metabolic dysfunction mediates the accumulation of liver fat and causes the continued development of MAFLD

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Evaluation of heme oxygenase-1 as a systemic biological marker of sporadic AD

Alternative buffering agents or careful titration of bicarbonate should therefore be considered, reserving therapy for situations where severe, life-threatening acidemia outweighs theoretical risks