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Under hypoxic conditions, human macrophages inhibit ferritin autophagy by reducing the expression of nuclear receptor coactivator 4 (NCOA4), resulting in increased mitochondrial ferritin expression and decreased intracellular free iron, thus regulating ferroptosis [35]

This rigorous methodology minimizes bias and allows for accurate attribution of adverse effects to the intervention rather than background health conditions or placebo effects

Inhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload

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food choices were left to the discretion of each participant

Metallomics 8, 597604