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Therefore, these findings underscore FSKs potential as a preventive and therapeutic agent for PD via the AC/cAMP/CREB pathway activation

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USP22 also deubiquitinates RIPK3, which causes high hyperglycemia to trigger damage to podocyte intercellular collagenization while targeting USP22 increased the amount of ubiquitinated degradation of RIPK3 and protected podocytes[97]

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BACH1 and Diabetes Diabetes is a chronic metabolic disorder known by a persistent state of high glucose in the blood, influenced by various genetic and environmental factors that impact the pancreatic cells function and insulin signaling pathways