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Indeed, peripheral ghrelin decreases afferent vagal discharge in the NTS whereas CCK increases discharge, suggesting that vagal stimulation of NTS targets represents an integration of orexigenic and anorexigenic signals (35, 36)

(2013), and Yan et al

These HMOs affect pathogen-associated molecular pattern signaling pathways [Toll-like receptor (TLR) 3, TLR5, IL-1, or cluster of differentiation 14 (CD14) expression and binding] decreasing cytokine production and the inflammatory response (56, 57)