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An increasing body of preclinical and clinical evidence suggests that mitochondrial dysfunction, characterized by bioenergetic failure, heightened oxidative stress, dysregulated calcium handling, and impaired mitophagy, contributes to the etiopathogenesis of both CVDs and cognitive impairment

Journal of Industrial Microbiology & Biotechnology 19

Fibrotic lung disease alters neutrophil trafficking and promotes neutrophil elastase and extracellular trap release

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The regulation of TG storage, lipolysis, and the subsequent release of fatty acids constitutes the core functions of PLINs

384,385 Such polarization enables the efficient translation of cytoskeletal forces into directed cell-body displacement