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[] Mitochondrial fusion necessitates tightly coordinated sequential merging of the outer and inner membranes, with inner mitochondrial membrane (IMM) fusion representing a critical regulatory checkpoint whose failure following successful OMM fusion triggers immediate organellar fission and fragmentation

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Accordingly, despite the loss of highly glycosylated proIGF-1Ea after the 2-DG treatment, the condition media of these cells fully activated the IGF-1R of human breast cancer cell line MCF-7 (Fig