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In the latter conformation, the rAmy bypass motif becomes disordered, enabling the receptor ECD to adopt a position similar to CT-bound CTR or AMYR complexes 15

These COX10-deficient oligodendrocytes maintained axonal thickness without a change in the number of oligodendrocyte progenitor cells (OPC) or astrocytes and did not induce microglial activation, suggesting that oligodendrocytes can function without oxidative phosphorylation

One proposal is that hypofunction of NMDA receptors (NMDAR) located on inhibitory GABA inter-neurons leads to reduced activity of these inter-neurons, resulting in increased pyramidal glutamatergic neurotransmission and the diverse symptoms occurring in acute psychosis [3]

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Sekhar RV, et al

p = ns)