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Cobalamin uptake and reactivation occurs through specific protein interactions in the methionine synthase-methionine synthase reductase complex

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One of the interactions they identified involved the cytosolic form of the selenoprotein glutathione peroxidase, GPX1, which bound strongly to the inactive C145A M pro mutant

Abstract The activation of endothelial cells (ECs) is a crucial step on the road map of tumor angiogenesis and expanding evidence indicates that a pro-oxidant tumor microenvironment, conditioned by cancer metabolic rewiring, is a relevant controller of this process